Tucker Carlson Interviews Dr. Peter McCullough on COVID Worldwide Conspiracy

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Of course, the word conspiracy is not used.  Professional people do not use such a word.  And we are all professionals here and everything is under control.  Well, actually, everything is not under control and something is “very very wrong” but we should not think to be deeply about such things, right?

Basically Doctor Peter McCullough goes into great lengths explaining how there are many treatments for Covid but that they are never mentioned and even actively suppressed. Both Doctor Peter McCullough and Tucker are trying to wrap their minds around this and seem to be totally baffled on how such treatments can be ignored.  There is too much in this interview to sum up here and it is worth watching in whole.

 

 

The is one thing I have been meaning to mention and it was brought up in this interview. The Lancet, the most prestigious medical journal in the world, published a paper that claimed the hydroxychloroquine is deadly.  This turned out to be completely false. In addition to this medicine being used very successfully for 40 years, the sources for the paper were obviously fraudulent.  For those that might not be familiar with academic writing, this is a huge deal. This simply does not happen, and was obviously published fraudulently on purpose.   It was retracted a couple weeks later but the world wide damage had already been done and the media never covered the retraction.  This one incident alone should set off the alarm bells.  For the most prestigious academic journal in the world to outright lie and potentially lead to millions of deaths from a deadly virus, the term “mind blowing” does not even come close to doing justice to what this information implies.  I have to emphasize again.  This is an academic journal, with editors and peer review.  Most people get rejected for the smallest of errors. To get published at all is an incredible feat and the level of scientific rigor of such a journal is always beyond reproach. Well, not this time apparently.  Right when humanity needed an objective medical opinion to what was to be done, (with what was the thought to be the one of the most dangerous plagues in modern history) what did humanity get from the medical elite?  Lies. What does such a lie imply?  I don’t know if words can do such a crime justice.  Purposeful mass killing of millions.  What is the word for that? I will let the reader figure that out.  More concerning in my mind, what comes next?

227,254 thoughts on “Tucker Carlson Interviews Dr. Peter McCullough on COVID Worldwide Conspiracy”

  8. Tesofensine is a prevention of noradrenaline, dopamine and serotonin reuptake that is also reported to indirectly boost the cholinergic system (Thatte, 2001) although the complete information of its medicinal profile are not extensively offered.

    An even more complete metabolic and genetic characterization in mix with thorough illness aetiology and reaction to various devices in drug activity need to result in an improvement in client treatment. Furthermore, this can additionally possibly cultivate the future generation of AOMs by advancing a deeper understanding right into the molecular pharmacology of body weight policy. It remains to be determined whether one, 2 or even more mechanisms in medication action will certainly prove effective in treatment of many clients with weight problems, or whether far more varied modification will certainly be needed to ideally tackle the obesity pandemic. The Dietary Supplement Health and Education Act (DSHEA) was accepted inthe United States in 1994, classifying dietary supplements as foods if they hadbeen in the food supply before 1994.

    The Anorexigenic Impacts Of Tesofensine Are Enhanced By The Chemogenetic Inhibition Of Lh Gabaergic Nerve Cells
    UCP1, localized in the inner mitochondrial membrane of brownish and off-white adipocytes, catalyses the transportation of protons across the mitochondrial membrane layer and, consequently, induces mitochondrial uncoupling of oxygen intake from ATP synthesis258,259. Pharmacologically, UCP1 task can be caused by catecholamines with subsequent activation of β3-adrenergic receptors of brownish adipose tissue257. Thyroid hormonal agent (T3) is an endogenous entity with uncoupling capability mediated by a number of various mechanisms260. Glucagon-like peptide 1 receptor (GLP1R) agonism applies both direct and indirect results on power and sugar metabolism in key outer organs as well as the brain.
    Medicinal Support For The Therapy Of Obesity– Existing And Future
    The stage I scientific trial with TM38837 was efficiently completed in 2009 (J.M. van Gerver, unpublished results). Orlistat is typically well endured; however, because of the non-absorbed fats in the intestine, individuals can experience steatorrhea, regular defecation, flatus with discharge, and fecal urinary incontinence. By co-prescribing a fiber-containing supplement, such as psyllium, the stomach adverse effects of orlistat can be decreased. As orlistat prevents the lipid-soluble vitamins from being absorbed, vitamin A, D, E, and K supplements need to be considered for long-lasting use. As a non-central nerves agent, orlistat inhibits the action of intestinal and pancreatic lipases, thereby blocking the hydrolysis of triglycerides and absorption of fatty acids carried out by the digestive endothelium.

    As expected, in Lean ChR2 mice, optogenetic activation of LH GABAergic nerve cells activated a binge in sucrose intake (Fig 5C, see blue line). Remarkably, at both doses, tesofensine successfully suppressed this feeding reaction, dramatically lowering cumulative licks contrasted to saline (Fig 5C and 5D5D, see #). These searchings for display the anorexigenic possibility of tesofensine in modulating LH GABA-driven feeding.

    Conditioned preference hostility was analyzed in beloranib-treated OLETF rats as a prospective system underlying reductions in food consumption (Kim et al., 2007a). Compared to car control, solitary peripheral shot of the favorable control, lithium chloride (0.15 M; vol was 2% body weight) and beloranib (1 or 10 mg/kg) created conditioned taste aversion (reduced saccharin solution consumption) in OLETF rats. The anorexigenic result of beloranib can be explained partly by the induction of preference hostility.
    For histological confirmation of electrode location in the mind, the electrodes were covered with DiI lipophilic carbocyanine color (1%; Sigma-Aldrich) permitting the monitoring of the fluorescent track left by the electrodes. Microsomal transfer protein is a heteromeric healthy protein involved in the synthesis of chylomicrons and apolipoprotein B-containing lipoproteins, influencing the transportation of lipids and cholesterol from the intestine and liver to tissues (Cuchel & Rader, 2013). First-generation microsomal transfer healthy protein preventions were developed to prevent hepatic healthy proteins and give an unique treatment for dyslipidemia (Roevens et al., 1999). While powerful preventions of hepatic microsomal transfer protein took in decreasing low-density lipoprotein-cholesterol, these preventions resulted in elevation of liver enzymes and hepatic steatosis in pets and people (Roevens et al., 1999; Gruetzmann et al., 2000). In the interesting and consistent look for enhanced anti-obesity medicines a wide array of representatives are and will be under examination as noted in Table 27. The search targets neuroendocrine peptide hormonal agents (vida supra), sirtuins, vaccines, over-the-counter agents, traditional natural plants and others.178,305,368 Several of these prospective chemicals are thought about currently.

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